There are 100 trillion cells in the human body and they are part of a highly organized community which regulates the total number of cells through control over the rate of cell division as well as cell death. When cells are no longer needed or have a threat to an organism, they will go through a suicidal program known as cell death or apoptosis. This process contains a series of special protein hydrolysis reactions, which will cause cell shrinkage and condensation, cytoskeleton decomposition, and cell surface change, and this in turns allows nearby phagocytes, such as macrophages, to adhere to the cell membrane and digest the cell. In contrary to programmed cell death, cell death which results from acute cellular injury usually loses cell membrane integrity and therefore causes the cell membrane to expand and break. This process is known as necrosis. Necrotic cells may release their cellular contents into the extracellular space and therefore induce inflammation and damage adjacent cells. However, apoptosis is an orderly cell death, which will induce cell decomposition and phagocytosis before any release of cellular components occurs and thus the adjacent cells usually still maintain normal. Apoptosis is initiated through the activation of a family of proteases called by caspases. These enzymes are synthesized and stored in the cell in the form of inactive procaspases. The mechanism to activate caspases is very complex; however, once activated, these enzymes will split and activate the other procaspases, initiating a series of reactions which will rapidly decompose cellular proteins. Therefore, the cell can go through autolysis and the residues can quickly be decomposed by the nearby phagocytes. Tissues in the process of development and recombination will experience extremely large amount of apoptosis, and even in adults, about billions of cells in the tissues will die every hour, such as in the intestines and bone marrow, and are replaced by new cells. However, in healthy adults, programmed cell death and new cell formation should reach a precise balance, otherwise the body tissue will shrink or overgrow. The New Human Line can utilize the newly-discovered Absolutely Constant Energy Source (ACES) and the new biological engineering techniques to change ions in the cell, such as the concentration of sodium ions and chloride ions, and, by means of changes in the concentration, control the balance of ions in the cell and activate the procaspases which are originally stored in the cell. These enzymes, once activated, will split and activate the other procaspases, initiating a series of reactions which will rapidly decompose cellular proteins to have the cell go through autolysis and make the residues quickly decomposed by the nearby phagocytes so as to reach the purpose of rapid apoptosis. The evolutionary emergence of this new physiological function allows the New Human Line to voluntarily control the cycle of cell death. The purpose of this experiment is to prove that Mr. Yuan Lin, who is the first successfully-evolved New Human Line, can employ the function of activating apoptosis to activate chloride ions and change their concentration, and also, by means of changes in the concentration, to control the balance of chloride ions in the human body cells so as to initiate the mechanism of apoptosis and induce rapid cell death without any change in the molecular weight, structural formula, and conformation, at 25 ℃, 1.0 atm, and pH 7.0, in a confined and isolated space, and with no contact with catalysts, biologically active substances, chemical substances, and physical action forces. Table 1. Comparison of the amount of precipitate between two treatments From the above experimental data, it shows a significant difference (p = 0.04) in the concentration between the control samples and the samples treated with ACES by Mr. Yuan Lin. It verifies that Mr. Yuan Lin, the New Human Line, can employ the function of activating apoptosis to activate chloride ions and change their concentration so as to rapidly initiate the mechanism of apoptosis and induce cell death.
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