The Abilities of DNA Mobility and Modification of Genetic Defect for the Sensory Humans

The DNA sequences in the Sensory Humans are the foundation of biological heredity, development, and functional operation, including appearances of height, hair color, skin color, and eyes color; their IQ of abilities and innate talent as music and art and body diseases from genetic defect that are popular genetic diseases in Homo sapiens.

There are many factors to cause genetic diseases, including single gene defect, abnormal chromosome, multiple genes common function, and mitochondrial gene mutation. There are general concepts of common genetic diseases as following:

Glucose-6-Phosphate Dehydrogenase deficiency: abnormal low level of G-6-P-D, it is danger to hemolysis when we eat fava beans. When use quinine the hemolysis be happened too.
Cystic Fibrosis: abnormal protein leads to thick, viscous secretions in lungs and other parts of body.
Phenylketonuria: metabolic disorder phenylalanine hydroxylase caused problems with brain development.
Alpha-1 antitrypsin deficiency, AAT deficiency: when white blood cells destroy bacteria attack and normal protein can inhibit enzyme which released from white blood cells. Most of these patients cannot inhibit enzyme and cause emphysema.
Sickle Cell Anemia: red blood cells assume an abnormal, rigid, and sickle shape; it will be hemolysis in small blood vessels when it contains low oxygen.
Adenosine deaminase deficiency, ADA deficiency: affect normal functions of lymphocyte and immunity.
Duchenne muscular dystrophy: abnormal muscular proteins lead muscular degeneration and lose functions of muscle to arise difficult breath.
Hemophilia A: disorder protein is functional plasma clotting VIII which is necessary factor for blood clotting.
Tay-Sachs disease: disorder of enzyme that decomposes lipid of cell membrane; abnormal fragments of cell membrane accumulate to lead nervous system degeneration.
Colorblind: it is a sex-linked genetic recessive disease, patient is completely inability to distinguish colors; they have other problems with eyes as poor vision, nystagmus, photosensitive reaction, and extra poor vision.
Familial hypercholesterolemia, FHC: affect absorption of cholesterol and cause the hardened blood vessels and coronary artery diseases.
Huntington’s disease: neurodegenerative genetic disorder disease
X-linked hypophosphatemic rickets: mutations in PHEX protein in kidney lead the low concentration of phosphate in the blood and let parathyroid hormone long stimulate bones to release calcium ion and phosphate and cause fragile and deformed bone.
Thalassemia: disorder or decrease of hemoglobin synthesis in red blood cells.
Down’s syndrome: unbalance copy of chromosome causes mild or serious disabilities of congenital ability and many changes of physical growth.

In the recent researches, there are many cell, tissue, organ, and system diseases in human body, for examples: coronary heart diseases, high blood pressure, stroke, diabetes, and cancers etc. and mutation mitochondrial genes almost relate to genetic diseases.

These popular genetic diseases in Homo sapiens, not only they are impossible to repair but also difficult to keep in normal condition even though they are limited effective through childbirth screening. The main reason is that DNA sequences of Homo sapiens are short of functions which can freely mobile and modify genetic DNA damage and lead genes incapable of repairing the damage DNA fragments during gene transcription and translation. It made damage DNA fragments inherit to next generation.

DNA sequences in the Sensory Humans has a function which can freely mobile and modify genetic DNA damage and repair the damage DNA fragments by using mobility DNA under transcription and translation to keep normal condition. Also it can use mobility to modify amino acid sequences and peptide formation processes, when the produce protein signals of DNA fragments happened mutation, it will produce abnormal functions of protein or it will not produce enough quantity normal protein and lead abnormal genetic appearance. This evolutionary function of mobility modification of genetic DNA damage appeared and let the Sensory Humans resist the genetic diseases attack forever and also completely prevent pain and scare from genetic diseases.

The abilities of DNA mobility and modification of DNA damage for the Sensory Humans make the Sensory Humans capable of modifying genetic DNA damage and avoid transmission and spread genetic diseases in ethnic group.

Mr. Yuan Lin who is the first Sensory Humans, he can utilize mobility DNA to modify genetic DNA damage. The result of experiment was shown as following:

The ultrasound was taken during third months of embryo development. It showed clearly that no genetic diseases and strong chordate in embryo of second generation of the Sensory Humans. It was through the Sensory Humans to mobilize DNA and modify genetic DNA damage.

※This paper is a simple edition on website. The formal edition contains the research theories and techniques, and is classified information. The information provides only to the members of Chinese Association for the Human Evolution and specific responsible members of specialized committees for the purpose of academic research whenever it is decrypted.

 (Copyright of this article is reserved by Mr. Yuan Lin. This article cannot be transcribed or reprinted without the permission.)